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Massimo Cristofanilli

Affiliation : NewYork–Presbyterian Hospital · Weill Cornell Medicine

• 3Publications (affiliation in the United States)
• 36Total publications
• 117Citations
• 4h-index

OpenAlex

Research profile

Massimo Cristofanilli has 3 indexed publications (2024–2026). Their work appears mainly in “Clinical Cancer Research”, “Cancer Cell International” and “Annals of Oncology”. Their most cited article, “Quantitative HER2 profiling on circulating tumor cells using an EpCAM-independent platform in metastatic breast cancer” (2025), has been cited 4 times. Their work is co-authored with at least 12 researchers, including institutions in Italy, Tunisia.

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Research areas

• Breast Cancer Treatment Studies
• Cancer Genomics and Diagnostics
• Advanced Breast Cancer Therapies
• BRCA gene mutations in cancer
• Cancer Cells and Metastasis

Publications (3)

• Abstract PS2-12-18: The role of MARCKS in inflammation in inflammatory breast cancer using in vitro and in silico studies
  2026 · Clinical Cancer Research
• Quantitative HER2 profiling on circulating tumor cells using an EpCAM-independent platform in metastatic breast cancer
  2025 · Cancer Cell International · cited 4 times
• 415P Comprehensive liquid biopsy characterization of patients with metastatic inflammatory breast cancer
  2024 · Annals of Oncology · cited 3 times

Publication venues

• Clinical Cancer Research × 1
• Cancer Cell International × 1
• Annals of Oncology × 1

Publications per year

• 20261
• 20251
• 20241

Collaborations

Partner institutions : Robert H. Lurie Comprehensive Cancer Center of Northwestern University · University of Udine · Cornell University · Università Cattolica del Sacro Cuore · Institut Pasteur de Tunis

Co-authors’ countries : Italy · Tunisia

Co-authors

• Nadia Bayou
• Laura Sofia Munoz-Arcos
• Mara Serena Serafini
• Eleonora Nicolò
• C. Reduzzi
• Sarah J. Henretta
• WJ Gradishar
• Huiping Liu
• Olivier Elemento
• Jeannine Donahue
• Carolina Reduzzi
• Elisabetta Molteni

Bibliographic data from OpenAlex (open database).